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PDF] Non-Small Cell Lung Cancer Cells Acquire Resistance to the ALK Inhibitor Alectinib by Activating Alternative Receptor Tyrosine Kinases. | Semantic Scholar
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Beyond EGFR, ALK and ROS1: Current evidence and future perspectives on newly targetable oncogenic drivers in lung adenocarcinoma - ScienceDirect
Met – Wikipedia
TargetingOne Corporation., Ltd
Activated MET acts as a salvage signal after treatment with alectinib, a selective ALK inhibitor, in ALK-positive non-small cell lung cancer
Inhibitory role of Alectinib on EML4-ALK positive NSCLC and activation... | Download Scientific Diagram
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Cancers | Free Full-Text | Molecular Characteristics of Radon Associated Lung Cancer Highlights MET Alterations
Biomarkers in Lung Cancer: Integration with Radiogenomics Data | IntechOpen
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Crizotinib in advanced non-small-cell lung cancer with concomitant ALK rearrangement and c-Met overexpression | BMC Cancer | Full Text
Metformin reduces HGF-induced resistance to alectinib via the inhibition of Gab1 | Cell Death & Disease
Expression levels of ROS1/ALK/c-MET and therapeutic efficacy of cetuximab plus chemotherapy in advanced biliary tract cancer | Scientific Reports
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Frontiers | Prevalence and Clinical Impact of Concomitant Mutations in Anaplastic Lymphoma Kinase Rearrangement Advanced Non-small-Cell Lung Cancer (Guangdong Association of Thoracic Oncology Study 1055)
Coexistent genetic alterations involving ALK, RET, ROS1 or MET in 15 cases of lung adenocarcinoma | Modern Pathology
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Crizotinib is an Orally Active, ATP-Competitive ALK and c-Met inhibitor - Network of Cancer Research