Home

Lotterie aus Archäologie thapsigargin er stress mechanism Rund und rund bedingungslos Vergeltung

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text

Biomedicines | Free Full-Text | The Crosstalk of Endoplasmic Reticulum (ER)  Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer,  Inflammation and Infection
Biomedicines | Free Full-Text | The Crosstalk of Endoplasmic Reticulum (ER) Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer, Inflammation and Infection

HRC promotes anoikis resistance and metastasis by suppressing endoplasmic  reticulum stress in hepatocellular carcinoma
HRC promotes anoikis resistance and metastasis by suppressing endoplasmic reticulum stress in hepatocellular carcinoma

Effect of endoplasmic reticulum stress on metabolic and stress signaling  and kidney-specific functions in Madin-Darby bovine kid
Effect of endoplasmic reticulum stress on metabolic and stress signaling and kidney-specific functions in Madin-Darby bovine kid

Tunicamycin specifically aggravates ER stress and overcomes chemoresistance  in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation |  Journal of Experimental & Clinical Cancer Research | Full Text
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text

MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria  contact sites | The EMBO Journal
MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria contact sites | The EMBO Journal

Schematic diagram of thapsigargin-stimulated TRAIL-induced apoptosis in...  | Download Scientific Diagram
Schematic diagram of thapsigargin-stimulated TRAIL-induced apoptosis in... | Download Scientific Diagram

ER stress-induced cell death mechanisms - ScienceDirect
ER stress-induced cell death mechanisms - ScienceDirect

Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs
Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs

Thapsigargin-induced severe ER stress stimulates release of... | Download  Scientific Diagram
Thapsigargin-induced severe ER stress stimulates release of... | Download Scientific Diagram

EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download  Scientific Diagram
EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download Scientific Diagram

Targeting endoplasmic reticulum stress in insulin resistance: Trends in  Endocrinology & Metabolism
Targeting endoplasmic reticulum stress in insulin resistance: Trends in Endocrinology & Metabolism

Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in  Pharmacological Sciences
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences

Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau  Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin -  2021 - Angewandte Chemie International Edition - Wiley Online Library
Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin - 2021 - Angewandte Chemie International Edition - Wiley Online Library

Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA  Translation of the p53 Isoform p53/47 - ScienceDirect
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect

Revealing Protein Aggregates under Thapsigargin-Induced ER Stress Using an  ER-Targeted Thioflavin | ACS Sensors
Revealing Protein Aggregates under Thapsigargin-Induced ER Stress Using an ER-Targeted Thioflavin | ACS Sensors

Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia

Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in  Pharmacological Sciences
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences

Endoplasmic reticulum stress signalling – from basic mechanisms to clinical  applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library
Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library

Endoplasmic reticulum stress stimulates the release of extracellular  vesicles carrying danger-associated molecular pattern (DAMP) molecules |  Oncotarget
Endoplasmic reticulum stress stimulates the release of extracellular vesicles carrying danger-associated molecular pattern (DAMP) molecules | Oncotarget

Effects of endoplasmic reticulum stress on apoptosis induction in  radioresistant macrophages
Effects of endoplasmic reticulum stress on apoptosis induction in radioresistant macrophages

Thapsigargin - Wikipedia
Thapsigargin - Wikipedia

IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to  Anticancer Drug
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug

Endoplasmic reticulum stress, the unfolded protein response and autophagy  in kidney diseases | Nature Reviews Nephrology
Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases | Nature Reviews Nephrology

ER stress-induced cell death proceeds independently of the TRAIL-R2  signaling axis in pancreatic β cells | Cell Death Discovery
ER stress-induced cell death proceeds independently of the TRAIL-R2 signaling axis in pancreatic β cells | Cell Death Discovery

Frontiers | The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and  Neurodegeneration in Parkinson's Disease
Frontiers | The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and Neurodegeneration in Parkinson's Disease

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text